Podcasts by Charles Ortleb

Here is a list of Rebecca Culshaw Smith's 20 most important points about HIV and AIDS

Rebecca Culshaw Smith’s Substack, “The Real AIDS Epidemic,” highlights core criticisms of mainstream HIV/AIDS theory, medical testing, pharmaceutical practices, and challenges to scientific orthodoxy. Based on her popular posts, interviews, and thematic content, these are 20 of the most important ideas advanced on her platform:

1. Questioning the existence of HIV as a unique virus, arguing that classic virological isolation (Koch’s postulates) has not been fulfilled.

2. Highlighting the non-specificity and cross-reactivity of HIV antibody tests, leading to potential misdiagnosis.

3. Criticism of “viral load” PCR tests for not detecting whole pathogens but only RNA fragments.

4. Noting the shifting criteria for HIV test positivity over time, calling diagnostic standards into question.

5. Documenting long-term “non-progressors” and “elite controllers” who remain healthy without antiretroviral therapy.

6. Raising awareness of AIDS-defining illnesses in HIV-negative individuals and questioning causality.

7. Arguing that hazard from AIDS medications (e.g., AZT, Truvada, Prep) may outweigh their benefits, especially due to their toxicity and inconsistent trial results.

8. Critique of the marketing and deployment of pre-exposure prophylaxis (Prep), calling it a pharmaceutical “scandal” targeting people not at significant risk.

9.Exploring how COVID-19 public health narratives mirror what she views as deception and fear tactics from the AIDS era.

10. Disputing the epidemiological narrative that AIDS is globally caused by a single infectious agent, and highlighting massive regional/demographic inconsistencies.

11. Exposing groupthink, censorship, and reputational shaming used against scientists questioning the HIV/AIDS paradigm.

12. Emphasizing failures of antiretroviral therapy in preventing disease progression for many patients.

13.. Explaining the statistical and mathematical problems in foundational HIV/AIDS research and the “shaky foundation” of guiding studies.

14. Arguing that AIDS-defining diseases may often reflect toxicity, malnutrition, or existing comorbidities, not a distinct viral syndrome.

15. Linking historical and social factors (such as drug use, pharmaceutical incentives) to the creation and persistence of the HIV/AIDS establishment.

16. Alerting readers to issues of false positive antenatal screening and broader concerns about mass diagnostic testing in medicine.

17. Suggesting that “virus-like particles” in the body  are misidentified as pathogens, not proof of HIV’s existence.

18. Forecasting that advances in AI and technology may help overturn scientific “consensus” by increasing transparency and debate.

19 Publicly refuting hit pieces and attempts to “cancel” her work as ideological suppression, not science.

20.Advocating for a return to fundamental scientific rigor and genuine skepticism in medical research, especially around virology and public health narratives.

These topics synthesize her core objections to HIV/AIDS orthodoxy and frame her Substack as a point of dissent and critique against modern medical paradigms and their social consequences.

K.S. and AIDS seem not to be two different epidemics.

From Perplexity A.I.

 

A close look at the facts about Kaposi's Sarcoma may cause a major shift in the AIDS PARADIGM.

If autopsy studies—such as those reported by George Hensley—found that nearly all AIDS patients show internal Kaposi's Sarcoma (KS) lesions, yet not all such patients test positive for HHV-8 (Human Herpesvirus 8), this discrepancy fundamentally challenges the prevailing theory that HHV-8 is the sole and necessary causal agent for KS in AIDS.

Evidence from Autopsy Studies
Hensley's team found KS-like lesions in about 95% of AIDS autopsies, far exceeding the clinical diagnosis rate and implying KS is almost universal internally among AIDS patients.

These autopsies revealed a broad morphologic spectrum and inflammatory variants of KS present in nearly all risk groups, independent of external symptoms.

HHV-8 Positivity is Not Universal
The scientific literature overwhelmingly associates HHV-8 with most cases of KS, detecting viral DNA in a majority of lesions.

However, case reports and pathology studies document KS-like tumors in HIV-positive patients that are negative for HHV-8 by immunohistochemistry—a finding dubbed “atypical spindled endothelial proliferation,” with classic histologic and clinical features still matching KS.

This existence of HHV-8-negative KS challenges the central paradigm tying KS causation strictly to HHV-8, especially when these lesions are indistinguishable from those found in HHV-8-positive patients.

Implications for the Role of HHV-8
If widespread KS lesions occur in AIDS patients regardless of HHV-8 status, it raises questions about the sufficiency and necessity of HHV-8 for KS pathogenesis.

The data suggest that either:

KS can arise independently of HHV-8, possibly due to immune dysfunction, chronic inflammation, or exposure to other infectious or environmental agents, or

Current HHV-8 detection methods may miss variants or low-level infections, or that HHV-8 acts in concert with other pathogenic factors, not alone.

This challenges therapeutic and diagnostic practices that rely solely on HHV-8 as a biomarker or causal link and opens a line of inquiry: Are there alternative mechanisms or cofactors in KS development—particularly in the unique immunological milieu of AIDS?

Overall, the autopsy data and reports of HHV-8 negative KS suggest that the role of HHV-8 in Kaposi's Sarcoma—especially in the context of AIDS—is not as clear-cut or exclusive as often assumed. This demands a re-examination of KS pathogenesis in immunosuppressed populations and a more nuanced model that allows for multiple contributing factors.

Why the mainstream media ignores Dr. Rebecca Culshaw Smith

Why the radio silence about Dr. Rebecca Culshaw Smith?

Perplexity A.I. explains.

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